Neural-renal interactions in the hypertension induced by papillary necrosis: Role of dietary salt intake

Keyphrases

• Hypertension 100%
• Dietary Salt Intake 100%
• Papillary Necrosis 100%
• 2-Bromoethylamine Hydrobromide 100%
• High Salt Intake 30%
• Dopamine 20%
• Salt Intake 20%
• Sodium Chloride 10%
• Lipids 10%
• Renal Function 10%
• Dopamine Level 10%
• Brainstem 10%
• Sympathetic Nervous System 10%
• Adrenergic Receptors 10%
• Central Nervous System 10%
• Binding Site 10%
• Serotonin 10%
• Drinking Water 10%
• Salt Load 10%
• Urinary Concentrating Mechanism 10%
• Necrosis 10%
• Renal Dopamine 10%
• Primary Defects 10%
• Glucose Excretion 10%
• Salt-induced 10%
• Induced Hypertension 10%
• Urinary Protein Excretion 10%
• Mild Hypertension 10%
• Drinking Tap Water 10%
• Creatinine Excretion 10%
• Neural Components 10%
• Hypertensive 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Sodium Chloride 100%
• 2 Bromoethylamine Hydrobromide 100%
• Kidney Papilla Necrosis 100%
• Noradrenalin 40%
• Binding Site 10%
• Adrenergic Receptor 10%
• Creatinine 10%
• Serotonin 10%
• Antihypertensive Agent 10%
• Tap Water 10%
• Borderline Hypertension 10%
• Necrosis 10%